№ files_lp_3_process_9_45736
Structured clinical and molecular case report form documenting hematological, biochemical and genetic findings in a patient with sickle cell disease caused by the HbS (HBB c.20A>T; p.Glu7Val) variant.
Year: Not specified
Country of origin: Surinam
Institution: The Hemoglobin University
Authors: Bento C; Traeger-Synodinos J; Kountouris P; et al.
Thematic area: Hematology; Hemoglobinopathies; Molecular genetics
Document type: Case report submission form
Gene: HBB
Protein variant: p.Glu7Val
cDNA variant: c.20A>T
Variant name: HbS
Database accession: HbVar ID 226; IthaID 824
Phenotype: Abnormal Doppler; organ failure due to vaso-occlusive crisis; skeletal dysplasia
Alpha genotype: -alpha3.7/-alpha3.7
Beta genotype: betaS/betaS
Age at evaluation: 56 years
Age at diagnosis: 2 months
Sex at birth: Female
Family history: Two sisters carriers of HbS
Clinical history: Hydroxyurea treatment from childhood until 54 years; uncomplicated pregnancy at 23 years; exchange transfusion started at 54 years
Hematological parameters before transfusion: Reported at age 53 years (Hb, MCV, MCH, RBC, Hct, Ret, RDW)
Iron status: Ferritin 35 µg/L
Blood smear: Target cells at normal iron indicative of alpha-thalassemia trait
Biochemical studies: Capillary electrophoresis (Sebia); HPLC (Trinity PHR)
Molecular studies: Sanger sequencing of exon 1 of HBB gene
Transfusion status: Exchange transfusion from age 54 years
Price: 8 / 10 USD
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